link o he full text
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2894986/
Arch Sex Behav. 2011 April; 40(2): 247–257.
Published online 2009 December 29. doi: 10.1007/s10508-009-9579-2
PMCID: PMC2894986
NIHMSID: NIHMS172372
A Further Assessment of Blanchard’s Typology of Homosexual Versus Non-Homosexual or Autogynephilic Gender Dysphoria
Larry Nuttbrock,corresponding author Walter Bockting, Mona Mason, Sel Hwahng, Andrew Rosenblum, Monica Macri, and
Measured categorically, 68.5%, 12.4%, 16.8%, and 2.1% of the 571 MTFs were classified as homosexual, heterosexual, bisexual, or asexual, respectively. Measured along the continuous scale of androphilia, 8.6%were low,19.1%were intermediate, and 71.8% were high. Measured along the continuous scale of gynephilia, 63.7% were low, 20.8% were intermediate, and 14.7% were high.
I was in error about the percent which seems to be 76%
Current scientific controversies: different treatment strategies section of this paper
http://www.ncbi.nlm.nih.gov/pmc/arti...0/?tool=pubmed
Dtsch Arztebl Int. 2008 November; 105(48): 834–841.
Published online 2008 November 28. doi: 10.3238/arztebl.2008.0834
All of the 21 patients who received a new diagnosis of GID in our clinic up to mid-2008 (aged 5 to 17; 12 boys, 9 girls) had psychopathological abnormalities that, in many cases, led to the diagnosis of additional psychiatric disorders. As a rule, there were also major psychopathological abnormalities in their parents. The "motive for switching" among the 15 adolescents in the group was mainly a rejected (egodystonic) homosexual orientation (see figure), the development of which would have been arrested by puberty-blocking treatments.
This would suggest otherwise
http://www.ncbi.nlm.nih.gov/pmc/arti...0/?tool=pubmed
Dtsch Arztebl Int. 2008 November; 105(48): 834–841.
Published online 2008 November 28. doi: 10.3238/arztebl.2008.0834
Neurobiological genetic research has not yet convincingly shown any predominant role for genetic or hormonal factors in the etiology of GID (1).
Even among children who manifest a major degree of discomfort with their own sex, including an aversion to their own genitalia (GID in the strict sense), only a minority go on to an irreversible development of transsexualism (6). Irreversibility of the manifestations, however, is considered to be an indispensable requirement before the diagnosis of transsexualism can be made, or any body-altering treatments initiated. In England and Canada, in accordance with this view, hormonal treatment or surgery is not recommended until the patient’s somatic and psychosexual development is complete.
. Though prospective studies are lacking, a consensus of opinion holds that gender identity disorders in children and adolescents are often associated with serious emotional and behavioral problems and with a high psychiatric comorbidity (1, 8)
Multiple publications have concerned a possible traumatic etiology of gender identity disorders (14) and an overlap of the psychopathological findings in GID with those of borderline personality disorder (15, e11, e12, e13), although there is some controversy on the latter point (16). A profound disturbance of the mother-child relationship can often be empirically demonstrated and is postulated to be a causative factor (e14).
Other authors, in line with psychoanalytic theory, do not attribute the desire to belong to the opposite sex to any prior trauma. Rather, they postulate the formation of a classic neurotic compromise, in which the child symbolically achieves a symbiotic fusion with the loved parent by switching genders
The irreversability is only diagnosed after one reaches puberty so it seems that one cannot be born a transsexual but one can be born with GID.